Epidermal growth factor activation of rat parotid gland adenylate cyclase and mediation by a GTP-binding regulatory protein
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Cited by (31)
Cellular signals underlying Β-adrenergic receptor mediated salivary gland enlargement
2012, DifferentiationCitation Excerpt :We previously reported isoproterenol-induced EGFR transactivation in a human salivary gland cell line (HSY), in which the functional β-AR population is exclusively of the β2 subtype (Klinger et al., 2002). EGF, like isoproterenol, is known to cause an increase in DNA synthesis and tissue enlargement in salivary glands (Nakagawa et al., 1991). We have demonstrated that EGF upregulates β2-AR expression and signaling in HSY cells (Yeh et al., 2003).
The juxtamembrane region of the epidermal growth factor receptor is required for phosphorylation of Gα(s)
2000, Archives of Biochemistry and BiophysicsTransmodulation of epidermal growth factor receptor function by cyclic AMP-dependent protein kinase
1999, Journal of Biological Chemistry[21] Epidermal growth factor-mediated regulation of G proteins and adenylylcyclase in cardiac muscle
1996, Methods in Neurosciences
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