DOI, a 5-HT2A/2C receptor agonist, potentiates amphetamine-induced dopamine release in rat striatum
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2021, Progress in Brain ResearchCitation Excerpt :This effect suggests the existence of 5-HT2A receptor-dependent inhibitory control upon cortical DA release, a 5-HT2C receptor-dependent effect (see below) or a 5-HT2A receptor-independent mechanism (Ichikawa et al., 2001b; Liegeois et al., 2002). Moreover, DOI, without an effect by itself, potentiated amphetamine or MDMA-induced DA efflux in the mPFC, NAc and striatum (Gudelsky et al., 1994; Ichikawa and Meltzer, 1995a; Kuroki et al., 2003). Compelling evidence from biochemical and electrophysiological studies indicates that 5-HT2A receptors may modulate the impulse flow-dependent and independent release of DA through mechanisms involving regulation of either DA synthesis or DA neuron firing rate (Gudelsky et al., 1994; Palfreyman et al., 1993; Schmidt and Fadayel, 1996; Schmidt et al., 1992b).
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