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Cited by (128)
Hypothalamic CRH neurons: A crossroad between stress and metabolism
2022, Current Opinion in Endocrine and Metabolic ResearchCitation Excerpt :Compelling evidence established that stress, or central administration of CRH, decreases food intake dose-dependently. This anorexigenic effect is potent and lasts 24 h or longer [26,27]. Consistent with this, CRH overexpression mice exhibit a reduction in fasting-induced food intake [28].
Physiologic and Neural Controls of Eating
2016, Gastroenterology Clinics of North AmericaWhen do we eat? Ingestive behavior, survival, and reproductive success
2013, Hormones and BehaviorCorticotropin-releasing factor (CRF) and neuropeptide Y (NPY): Effects on inhibitory transmission in central amygdala, and anxiety- & alcohol-related behaviors
2012, AlcoholCitation Excerpt :It is interesting that CRF and NPY show a high degree of neuroanatomical overlap and largely opposite behavioral profiles. For example, CRF promotes increases in anxiety-like behavior (Koob & Thatcher-Britton, 1985), increases in arousal (Koob et al., 1984), and decreases in feeding (Levine, Rogers, Kneip, Grace, & Morley, 1983), whereas NPY promotes decreases in anxiety-like behavior (Heilig et al., 1993), decreases in arousal (Heilig & Murison, 1987), and increases in feeding (Stanley & Leibowitz, 1984). As discussed below, alcohol-related behaviors exhibit heightened sensitivity to manipulation of brain CRF and NPY systems in individuals that are either alcohol-dependent, genetically vulnerable to consume high quantities of alcohol drinking, repeatedly cycled through periods of alcohol withdrawal, or innately.