Effects of cocaine on release and uptake of dopamine in vivo: Differentiation by mathematical modeling
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Cited by (41)
Serotonin (2C) receptor regulation of cocaine-induced conditioned place preference and locomotor sensitization
2013, Behavioural Brain ResearchCitation Excerpt :Although the effects of psychostimulants on brain dopamine systems are well recognized, the actions of cocaine on serotoninsystems are also important in its addictive properties. Substantial evidence shows that cocaine binds to dopamine transporters and blocks the reuptake of dopamine, leading to increased levels of extracellular dopamine [1–5]. However, cocaine also blocks the reuptake of other monoamines including serotonin [6–10].
Effects Of Psychostimulants On Neurotrophins. Implications For Psychostimulant-Induced Neurotoxicity
2009, International Review of NeurobiologyCitation Excerpt :Cocaine (benzoylmethyl ecgonine) is a crystalline tropane alkaloid obtained from the leaves of the coca plant. Cocaine causes an increase in DA by preventing the reuptake of newly released DA into striatal neurons via DAT (Heikkila et al., 1975; Nicolaysen and Justice, 1988). Chronic cocaine administration can lead to a number of persisting alterations in behavior such as the long-lasting sensitization to DA agonists (Robinson and Becker, 1986) and development of paranoid psychoses (Satel et al., 1991), as well as enduring alterations in brain chemistry, including alterations in a number of neurotransmitters and receptor populations (Kuhar and Pilotte, 1996).
Acute and chronic continuous methamphetamine have different long-term behavioral and neurochemical consequences
2005, Neurochemistry International