Biochemical and Biophysical Research Communications
Volume 144, Issue 1, 14 April 1987, Pages 438-446
Histamine-induced phosphoinositide metabolism in cultured human umbilical vein endothelial cells. Association with thromboxane and prostacyclin release
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Cited by (59)
Histamine-Induced Contraction and Relaxation of Placental Chorionic Plate Arteries
2007, PlacentaCitation Excerpt :Our data also indicate a significant contribution of the H1-receptor to chorionic plate artery relaxation by histamine; the initial rapid relaxation was significantly blunted but not abolished in the presence of famotidine and the maintained dilatation was only abolished completely in the presence of famotidine and mepyramine. This relaxation is likely to be via effects on the vascular endothelium; H1-receptor activation has been suggested to activate nitric oxide release and prostacyclin synthesis in vascular endothelial cells [29,30] via activation of IP3 and mobilization of intracellular calcium [31]. This involvement of the endothelium is supported by the effects of blockers of endothelial-dependent relaxation.
Histamine Influences the Blood-Spinal Cord and Brain Barriers Following Injuries to the Central Nervous System
2004, Blood-Spinal Cord and Brain Barriers in Health and DiseaseBiological activities and metabolism of the lysophosphoinositides and glycerophosphoinositols
2002, Biochimica et Biophysica Acta - Molecular and Cell Biology of LipidsInvolvement of protein kinase C in hypoxia-induced desensitization of the β-adrenergic system in human endothelial cells
1996, Biochemical and Biophysical Research Communications
Copyright © 1987 Published by Elsevier Inc.