Rapid communicationEffect of 7-nitroindazole on drug-priming reinstatement of d-methamphetamine-induced conditioned place preference
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Cited by (30)
Enhanced methamphetamine-induced conditioned place preference in risk-taking rats
2020, Behavioural Brain ResearchAdministration of activated glial condition medium in the nucleus accumbens extended extinction and intensified reinstatement of methamphetamine-induced conditioned place preference
2016, Brain Research BulletinCitation Excerpt :Many studies have documented that NO synthesis is involved in the expression of behavioral effects of METH and also, can affect mesolimbic dopamine system. It has been demonstrated that pretreatment with 7-nitroindazole, a NO inhibitor, attenuated the reinstatement of METH (Li et al., 2002a). Furthermore, the administration of 7-nitroindazole before METH treatment could significantly attenuate the acquisition and expression of METH-induced CPP (Li et al., 2002b).
Elevated BDNF mRNA expression in the medial prefrontal cortex after d-amphetamine reinstated conditioned place preference in rats
2014, NeuroscienceCitation Excerpt :Recent studies have applied CPP to test the reinstatement of drug, mostly for cocaine and morphine (Aguilar et al., 2009). Only a few studies reported on the reinstatements of drug CPP using AMP and its derivatives including d, l-AMP (Cruz et al., 2008, 2010), methamphetamine (Li et al., 2002; DeMarco et al., 2009; Qi et al., 2009; Do Couto et al., 2011; Yu et al., 2011; Abulseoud et al., 2012; Berry et al., 2012; Subiah et al., 2012) and 3,4-methylenedioxymethylamphetamine (MDMA) (Daza-Losada et al., 2011; Vidal-Infer et al., 2012). To our knowledge, the present work is the first in reporting drug-primed CPP reinstatement using d-AMP.
Role of the NO/sGC/PKG signaling pathway of hippocampal CA1 in morphine-induced reward memory
2012, Neurobiology of Learning and MemoryCitation Excerpt :Moreover, mice without the nNOS gene were resistant to cocaine-induced CPP (Itzhak et al., 1998). Similarly, 7-NI (12.5–50 mg/kg, i.p.) suppressed the acquisition, expression and reinstatement of D-methamphetamine induced CPP in rats (Li, Ren, & Zheng, 2002; Li, Yin, Shi, Lin, & Zheng, 2002). With respect to the role of NO in opiate-induced CPP, systemic injection of 7-NI and the non-selective NOS inhibitor L-NG-nitroarginine (L-NOARG) blocked morphine CPP in mice and rats (Kivastik, Rutkauskaite, & Zharkovsky, 1996; Manzanedo, Aguilar, Rodriguez-Arias, Navarro, & Minarro, 2004).