The Journal of Steroid Biochemistry and Molecular Biology
Volume 59, Issue 2, October 1996, Pages 233-240
Membrane binding sites and non-genomic effects of estrogen in cultured human preosteoclastic cells
References (34)
Estrogens in the prevention and treatment of postmenopausal osteoporosis: a review
Am. J. Med.
(1988)- et al.
Estradiol membrane binding sites on human breast cancer cell lines
Use of a fluorescent estradiol conjugate to demonstrate plasma membrane binding systems
J. Steroid Biochem.
(1986) - et al.
Steroid hormones regulate cAMP and cGMP production by procine granulosa, cells in vitro
J. Steroid Biochem. Molec. Biol.
(1993) - et al.
Effect of estrogen on acidification in osteoclasts
Biochem. Biophys. Res. Commun.
(1993) - et al.
Specific binding of estrogen to osteoclast surface
Biochem. Biophys. Res. Commun.
(1994) - et al.
Effects of endothelin-1 on bovine parathyroid cells
Biochem. Biophys. Res. Commun.
(1993) - et al.
A new generation of Ca2+ indicators with greatly improved fluorescence properties
J. Biol. Chem.
(1985) - et al.
Regulation of osteoclastic acid secretion by cGMP-dependent protein kinase
Biochem. Biophys. Res. Commun.
(1994) Local and systemic factors in the pathogenesis of osteoporosis
N. Engl. J. Med.
(1988)- et al.
Evidence of estrogen receptors in normal human osteoblast-like cells
Science
(1988)
Functional estrogen receptors in osteoblastic cells demonstrated by transfection with a reporter gene containing an estrogen response element
Molec. Endocr.
(1991)
Estrogen binding, receptor mRNA, and biological response in osteoblast-like osteosarcoma cells
Science
(1988)
Avian osteoclasts as estrogen target cells
Phorbol ester induced osteoclast-like differentiation of a novel human leukemic cell line (FLG 29.1)
J. Cell. Biol.
(1992)
Functional estrogen receptors in a human preosteoblastic cell line
Endometrial cell calcium and oestrogen action
Nature
(1975)
Metabolic and proliferative responses to estrogen by hepatocytes selected for plasma membrane binding-sites specific for estradiol-17
J. Cell. Physiol.
(1979)
Cited by (62)
Peripheral estradiol induces temporomandibular joint antinociception in rats by activating the nitric oxide/cyclic guanosine monophosphate signaling pathway
2009, NeuroscienceCitation Excerpt :The peripheral non-genomic antinociceptive effect of estradiol is mediated by the activation of the NO–cGMP signaling pathway, since it was blocked by the administration of the NO synthase inhibitor, l-NNA or of the guanylate cyclase inhibitor, ODQ in the ipisilateral TMJ. These data are in accordance with findings showing that estradiol increases the activity of the NO synthase (Chen et al., 1999; Simoncini et al., 2000) and guanylate cyclase (Fiorelli et al., 1996) through non-genomic mechanisms. The increased activity of these enzymes results in an increased level of cGMP, which has been associated with peripheral antinociception (Durate et al., 1990; Qian et al., 1996; Almeida and Duarte, 2007).
Regulation of Bone Cell Function by Estrogens
2008, Osteoporosis, Two-Volume SetEstrogens and Progestins
2008, Principles of Bone Biology: Volume 1-2, Third EditionNongenomic actions of low concentration estrogens and xenoestrogens on multiple tissues
2007, Molecular and Cellular EndocrinologyNeuronal differentiation of PC12 cells abolishes the expression of membrane androgen receptors
2006, Experimental Cell Research
Copyright © 1996 Published by Elsevier Ltd.