Abstract
The understanding of the pathophysiology of cerebral ischemic anoxia depends on elucidation of cellular neuronal changes during this event and under conditions of cardiopulmonary resuscitation. Current knowledge of these metabolic developments is reviewed, and a hypothesis of the role of iron in this situation is presented.
MeSH terms
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Animals
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Brain / metabolism
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Brain Ischemia / metabolism*
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Brain Ischemia / therapy
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Calcium / metabolism*
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Dogs
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Free Radicals
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Humans
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Hypoxia, Brain / metabolism*
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Hypoxia, Brain / therapy
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Iron / metabolism*
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Lipid Peroxides / metabolism
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Oxygen / metabolism
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Resuscitation*
Substances
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Free Radicals
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Lipid Peroxides
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Iron
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Oxygen
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Calcium