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- Published online March 26, 2009.
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- Earlier version (March 16, 2009 - 18:46).
- Earlier version (March 24, 2009 - 10:26).
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The American Society for Pharmacology and Experimental Therapeutics
Author Information
- Steven D. Buckingham,
- Andrew K. Jones,
- Laurence A. Brown, and
- David B. Sattelle
- Medical Research Council Functional Genomics Unit, Department of Physiology Anatomy and Genetics, University of Oxford, Oxford, United Kingdom
- Address correspondence to: Prof. David B. Sattelle, MRC Functional Genomics Unit, Department of Physiology Anatomy and Genetics, University of Oxford, South Parks Road, Oxford, UK, OX1 3QX. E-mail: david.sattelle{at}dpag.ox.ac.uk
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- Article
- Abstract
- I. Introduction
- II. β-Amyloid: Biophysics and Roles in Alzheimer's Disease
- III. Nicotinic Acetylcholine Receptors: Structure, Function and Roles in Neuroprotection
- IV. β-Amyloid Peptide Toxicity: Is It Mediated in Part through Nicotinic Acetylcholine Receptors?
- V. Loss of Cholinergic Neurons and Nicotinic Acetylcholine Receptors in Alzheimer's Disease
- VI. Nicotinic Acetylcholine Receptors: Molecular Switches Controlling Survival or Death
- VII. Future Directions
- VIII. Conclusion
- Acknowledgments
- Footnotes
- References
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