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- Article
- Abstract
- I. Introduction: Adolescent and Underage Drinking are Common But Differ From AUD
- II. Age of Drinking Onset and Risks for AUD
- III. Adolescent Intermittent Ethanol Exposure Procedure Models Human Underage Weekend Binge Drinking Exposure and Persistent Changes in Adulthood
- IV. Hippocampal Neurogenesis and the Neurogenic Niche: HMGB1 and Shifts in the Trophic/Proinflammatory Balance
- V. AIE and HMGB1 Signaling
- VI. Microglia, HMGB1, and Adolescent Alcohol Exposure
- VII. AIE Causes a Persistent Loss of Forebrain Cholinergic Neurons
- VIII. Reversal of AIE Pathology Identifies New Mechanisms and Potential Therapies
- IX. Summary and Conclusions
- Acknowledgments
- Authorship Contributions
- Footnotes
- Abbreviations
- References
- Figures & Data
- Info & Metrics
- eLetters
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