Regional distribution of methionine-enkephalin and substance P-like immunoreactivity in normal human brain and in Huntington's disease
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Cited by (244)
The Basal Ganglia
2012, The Human Nervous System, Third EditionGenetics and neuropathology of huntington's disease
2011, International Review of NeurobiologyCitation Excerpt :Similarly, by RIA Beal et al. (1988) observed extensive loss of SP from both SNr and SNc by Grade 1, followed by further loss in subsequent grades, with no clear differences between them at any grade. Other biochemical studies have reported varied results, however, with some observing greater loss of SP or GABA from SNr than SNc (Buck et al., 1981; Ellison et al., 1987; Emson et al., 1980; Kanazawa et al., 1977), and others the opposite (Gale et al., 1977). One study that distinguished HD cases as choreic (early to mid-HD) versus rigid (late HD) reported greater loss of GAD from SNr than SNc in both (Spokes, 1980).
Neurotransmission, oxidative stress, and coexistence of neurotransmitters in parkinson's disease
2007, Oxidative Stress and Neurodegenerative DisordersA histochemical and immunohistochemical analysis of the subependymal layer in the normal and Huntington's disease brain
2005, Journal of Chemical NeuroanatomyDifferential loss of striatal projection systems in Huntington's disease: A quantitative immunohistochemical study
2004, Journal of Chemical NeuroanatomyNeurotoxicity, Oxidative Stress and Cerebrovascular Disorders
2004, NeuroToxicology
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Present address: Dept. of Neurology, The Johns Hopkins Hospital, Baltimore, Md. 21205, U.S.A.
Copyright © 1980 Published by Elsevier B.V.