Bicuculline-insensitive gaba receptors on peripheral autonomic nerve terminals
References (49)
- et al.
Central effects of β-(p-chlorophenyl)-γ-aminobutyric acid
Brain Res.
(1974) - et al.
The action of β-phenyl-GABA derivatives on neurones of the cat cerebral cortex
Brain Res.
(1974) - et al.
A model for the mode of action of GABA on primary afferent terminals: depolarizing effects of GABA applied iontophoretically to neurones of mammalian dorsal root ganglia
Neuropharmacology
(1974) - et al.
Is Lioresal (baclofen) an antagonist of substance P?
Brain Res.
(1976) - et al.
Action of baclofen on mammalian synaptic transmission
Neuroscience
(1978) - et al.
Preferential metabolism of (−)-[3H]norepinephrine through the deaminated glycol in the rat vas deferens
Biochem. Pharmac.
(1973) - et al.
Uptake and metabolism of γ-aminobutyric acid by neurones and glial cells
Biochem. Pharmacol.
(1975) - et al.
Actions of GABA, picrotoxin and bicuculline on adrenal medulla
European J. Pharmacol.
(1974) - et al.
In vitro profile of some opioid pentapeptide analogues
European J. Pharmacol.
(1978) - et al.
Actions of γ-aminobutyric acid on sympathetic ganglion cells
J. Physiol. London
(1975)
A study of the factors affecting the aluminum oxide-trihydroxy-indole procedure for the analysis of catecholamines
J. Pharmac. Exp. Ther.
(1962)
Central depressant action of baclofen
J. Physiol. London
(1978)
γ-Aminobutyric acid uptake by sympathetic ganglia
Nature New Biol.
(1972)
Depolarizing actions of γ-aminobutyric acid and related compounds on rat superior cervical ganglia in vitro
Br. J. Pharmacol.
(1974)
[3H]-γ-Aminobutyric acid uptake into neuroglial cells of rat superior cervical sympathetic ganglia
J. Physiol. London
(1979)
Isoguvacine, isonipecotic acid, muscimol and N-methyl isoguvacine on the GABA receptor in rat sympatheric ganglia
Experientia
(1978)
Baclofen: a selective agonist for a novel type of GABA receptor
Br. J. Pharmacol.
(1979)
(−)-Baclofen decreases neurotransmitter release in the mammalian CNS by an action at a novel GABA receptor
Nature
(1980)
γ-Aminobutyric acid reduces the evoked release of [3H]-nor-adrenaline from sympathetic nerve terminals
Br. J. Pharmacol.
(1979)
A comparison of γ-aminobutyric acid and the semi-rigid analogues 4-aminotetrolic acid, 4-aminocrotonic acid and imiddazole-4-acetic acid on the isolated superior cervical ganglion of the rat
Br. J. Pharmacol.
(1976)
Inhibitors of neuronal GABA uptake potentiate the inhibitory effect of GABA on the field-stimulated guinea-pig vas deferens preparation
Br. J. Pharmacol.
(1981)
Partial agonists for brain GABA/benzodiazepine receptor complex
Nature
(1979)
Influence of neuroglial transport on the action of γ-aminobutyric acid on mammalian ganglion cells
Br. J. Pharmacol.
(1977)
Presynaptic effects of γ-aminobutyric acid in isolated rat superior cervical ganglia
Br. J. Pharmacol.
(1979)
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2017, Frontiers in NeuroendocrinologyCitation Excerpt :Given the central role of GABAergic neurotransmission in the SCN, a thorough characterization of the GABAA receptors within the SCN remains a large gap in our knowledge. The GABAB receptor was first identified more than three decades ago, after the demonstration that GABAA receptor antagonists were unable to block the late component of inhibitory transmission (Hill and Bowery, 1981; Bowery et al., 1981). GABAB receptors, like other G protein coupled receptors, have a central core domain composed of seven transmembrane helices.
Reflections on more than 30 years association with hanns
2015, Advances in PharmacologyNeurotransmitter Receptors in the Basal Ganglia
2010, Handbook of Behavioral Neuroscience
- ∗∗
Present address: MRC Neurochemical Pharmacology Unit, Hills Road, Cambridge, England.
Copyright © 1981 Published by Elsevier B.V.