Research paperApolipoprotein E-rich HDL in patients with homozygous familial hypercholesterolemia
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HDL functionality in familial hypercholesterolemia: effects of treatment modalities and pharmacological interventions
2018, Drug Discovery TodayCitation Excerpt :Several lines of evidence have shown that HDL2 particles can acquire apoE from arterial monocyte-derived macrophage foam cells and constitute apoE-containing HDL2 particles that can then be atherogenic [61]. The pool size of HDL-apoE in FH patients has been reported to be twofold higher than that in healthy subjects [62,63]. HDL-apoE has been proposed to bind to the extracellular proteoglycans, such as biglycan, and be retained in the arterial matrix of the atherosclerotic lesion [64–66].
LDL-apheresis depletes apoE-HDL and pre-β1-HDL in familial hypercholesterolemia: Relevance to atheroprotection
2011, Journal of Lipid ResearchCatabolism of HDL1 cholesteryl ester in the rat. Effect of ethinyl estradiol treatment
1999, Comptes Rendus de l'Academie des Sciences - Serie IIISeparation oa ApoA- and ApoB-containing lipoproteins of human plasma by affinity chromatography on concanavalin A
1991, Progress in Lipid Research
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