Regular articleAnaphylactic shock-induced conditioned taste aversion: II. Correlation between taste aversion and indicators of anaphylactic shock☆
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2017, Neurobiology of Learning and MemoryCitation Excerpt :Additionally, a number of studies aimed to investigate conditioning effects of asthma-like symptoms, or histamine release. Results impressively indicate that mast cell functions are also responsive to behavioral conditioning paradigms (Djuric, Markovic, Lazarevic, & Jankovic, 1988; Irie, Maeda, & Nagata, 2001; Irie, Nagata, & Endo, 2002, 2004). Moreover, in healthy human volunteers proof-of-principle studies have demonstrated that conditioning or learned placebo responses can modulate peripheral immune responses, such as lymphocyte functions and cytokine production (Ader & Cohen, 1975; Goebel, Meykadeh, Kou, Schedlowski, & Hengge, 2008; Sabbioni et al., 1997; Schedlowski & Pacheco-Lopez, 2010; Wirth et al., 2011), as well as neuroendocrine functions, like release of glucocorticoids and growth hormones (Benedetti et al., 2003).
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2010, Brain, Behavior, and ImmunityCitation Excerpt :More recently, a conditioned reduction in disease severity in mice with experimental autoimmune encephalomyelitis was reported following conditioning with saccharin taste (CS) and alpha lipoic acid (US) (Jones et al., 2008). A series of studies addressed behavioral conditioning of asthma-like symptoms, anaphylactic shock (Djuric et al., 1988; Noelpp and Noelpp-Eschenhagen, 1951a–c; Palermo-Neto and Guimarães, 2000) or histamine release (Irie et al., 2001, 2002, 2004; Peeke et al., 1987). These experiments indicate that especially mast cell function (Marone et al., 2003) is responsive to conditioning protocols.
Weaken taste-LPS association during endotoxin tolerance
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This work was supported by the Republic of Serbia Research Fund, Belgrade.