Cancer Cell
Volume 18, Issue 3, 14 September 2010, Pages 282-293
ArticleModulation of K-Ras-Dependent Lung Tumorigenesis by MicroRNA-21
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Graphical Abstract
Highlights
► miR-21 overexpression promotes tumorigenesis in a mouse model of NSCLC ► miR-21 deletion suppresses tumorigenesis in a mouse model of NSCLC ► miR-21 targets multiple suppressors of Ras signaling enhancing proliferation ► miR-21 suppresses apoptosis by targeting proapoptotic genes enhancing survival
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Present address: miRagen Therapeutics, Boulder, CO 80301-3695, USA
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