Regular ArticleCiprofloxacin Suppresses Cyp3a in Mouse Liver by Reducing Lithocholic Acid-producing Intestinal Flora
References (30)
- et al.
The constitutive androstane receptor and pregnane X receptor function coordinately to prevent bile acid-induced hepatotoxicity
J. Biol. Chem.
(2004) - et al.
FXR, a bile acid receptor and biological sensor
Trends Cardiovasc Med.
(2000) - et al.
Analysis of bile acids in conventional and germfree rats
J. Lipid Res.
(1976) - et al.
Enhancement of the 7 alpha-de-hydroxylase activity of a gram-positive intestinal anaerobe by Bacteroides and its significance in the 7-dehydroxylation of ursodeoxycholic acid
J. Lipid Res.
(1982) - et al.
Protein measurement with the Folin phenol reagent
J. Biol. Chem.
(1951) - et al.
Rapid identification of the species of the Bacteroides fragilis group by multiplex PCR assays using group- and species-specific primers
FEMS Microbiol. Lett.
(2003) - et al.
Protective role of hydroxysteroid sulfotransferase in lithocholic acid-induced liver toxicity
J. Biol. Chem.
(2003) - et al.
Lithocholic acid feeding induces segmental bile duct obstruction and destructive cholangitis in mice
Am. J. Pathol.
(2006) - et al.
Changes in biliary and fecal bile acids in mice after treatments with diosgenin and beta-sitosterol
J. Lipid Res.
(1984) - et al.
Pregnane X receptor is a target of farnesoid X receptor
J. Biol. Chem.
(2006)
Regulation and induction of CYP3A11, CYP3A1 3 and CYP3A25 in C57BL/6J mouse liver
Arch. Biochem. Biophys.
(2007)
A novel method in the induction of reliable experimental acute and chronic ulcerative colitis in mice
Gastroenterology.
(1990)
Intestinal flora induces the expression of Cyp3a in the mouse liver
Xenobiotica
(2009)
The nuclear receptor PXR is a lithocholic acid sensor that protects against liver toxicity
Proc. Natl. Acad. Sci. USA
(2001)
An essential role for nuclear receptors SXR/PXR in detoxification of cholestatic bile acids
Proc. Natl. Acad. Sci. USA
(2001)
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